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NMDA-MAPK-CREB pathway mediated affective pain”
  • 所属机构名称：复旦大学
  • 成果类型：会议
  • 相关项目：SNAP-25 相关蛋白(SIP30) 介导神经病理性疼痛的分子机制
同会议论文项目
SNAP-25 相关蛋白(SIP30) 介导神经病理性疼痛的分子机制
期刊论文 12 会议论文 13 著作 1
同项目会议论文
Involvement of ERK cascades in the Anterior Cingulate Cortex in Affective Pain.
NMDA receptor in the ACC contributes to affective pain.
病理性疼痛的神经生物学机制
The Glycine site of NMDA Receptors in the Anterior Cingulate Cortex Contributes to Pain-Conditioned
MAPK in the anterior cingulate cortex contributes to pain-related aversion
SIP30 involves in neuropathic pain in rats
Is Functional State of Spinal Glia Involved in the Anit-allodynic and Anti-hyperalgesic Effects of E
ERK信号转导通路参与大鼠前扣带皮层痛相关厌恶情绪的形成
痛情绪和痛记忆形成的细胞和分子机制
Is endogenous D-serine in the rostral anterior cingulated cortex necessary for pain-related negative
NMDAR-dependent AC-ERK-CREB signaling pathway in the anterior cingulate cortex contributes to pain-r
SIP30, a new MAPK downstream target in chronic constriction injuried neuropathic pain model in rats