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Up-regulation of divalent metal transporter 1 in 6-hydroxydopamine intoxication is IRE/IRP dependent
所属机构名称:青岛大学
会议名称:XVIII WFN World Congress on Parkinson’s Disease and Related Disorders.
成果类型:会议
相关项目:铁导致的alpha-突触核蛋白的聚集在帕金森病中的作用研究
作者:
Jiang H|Song N|Wang J|Xie J|Xu H|Zhang S|
同会议论文项目
铁导致的alpha-突触核蛋白的聚集在帕金森病中的作用研究
期刊论文 12
会议论文 21
同项目会议论文
Alpha-synuclein aggregation is involved in the iron toxicity to SK-N-SH neuroblastoma cells.
Iron and alpha-synuclein. Brain iron metabolism and neurodegenerative disease meeting.
Iron and Parkinson’s disease..
Ceruloplasmin is Involved in the Nigral Iron Accumulation of 6-OHDA Lesioned Rats.
IRP1 is activated by PKC-dependent phosphorylation in dopaminergic cell line MES23.5 cells.
Activation of ATP-sensitive potassium channels enhances DMT1-mediated ferrous iron uptake in SK-N-SH
.Iron metabolism and Parkinson’s disease
Role of alpha-synuclein aggregation and the Nrf2/ HO-1 pathway in iron-induced neurotoxicity.
Up-regulation of DMT1+IRE and down-regulation of FP1 are involved in 6-hydroxydopamine-induced neuro
Iron transporters mis-regulation contributes to the nigral iron accumulation in Parkinson’s di
Nifedipine reduces iron accumulation and dopamine neuron degeneration in the iron-overloaded substan
Role of alpha-synuclein aggregation in iron-induced neurotoxicity
Expression of iron-related proteins and alterations in iron content in the temporal cortex of parkin
Nifedipine reduces iron-overload induced dopamine neuron degeneration by inhibition of iron accumula
A novel protection by Rg1 on MPP+ induced iron accumulation by ROS and nuclear factor-kappaB signal
Ferroportin1 but not hephaestin contributes to iron accumulation in a cell model of Parkinson’
Mysregulation of iron in Parkinson's disease and its relation to the dopaminergic cell death.
Ceruloplasmin is invovled in the nigral iron accumulation of 6-OHDA lesiond rats.
Activation of ATP-sensitive potassium channels enhanced iron uptake and iron-induced oxidative stres
The role of ATP-sensitive potassium channels on DMT1-mediated ferrous iron uptake in SK-N-SH cells.