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C3aR is required for defense against kidney infection induced by uropathogenic Escherichia coli in m
所属机构名称:西安交通大学
会议名称:25th International Complement Workshop
时间:2014.10
成果类型:会议
相关项目:补体C5a受体在急性肾盂肾炎发生过程中的作用及其机制研究
同会议论文项目
补体C5a受体在急性肾盂肾炎发生过程中的作用及其机制研究
期刊论文 7
会议论文 7
同项目会议论文
Forskolin, a potent cyclic-AMP pathway activator,protect mice from acute pyelonephritis induced by u
Forskolin, a potent cyclic-AMP pathway activator, protect mice from acute pyelonephritis induced by
C5aR deficiency and C5aR blockade protect mice from acute pyelonephritis.
Blocking C5aR with C5aR antagonist protects mice from uropathogenic Escherichia coli-induced kidney
C5a-C5aR interaction promotes kidney infection through acting on both ranal and circulating cells
Blocking C5aR with C5aR antagonist protects mice from uropathogenic Escherichia coli-induced kidney