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Using the larvae nematode Caenorhabditis elegans to evaluate neurobehavioral toxicity to metallic sa
Using the nematode Caenorhabditis elegans as a model animal for assessing the toxicity induced by mi
Metallothioneins are required for formation of cross-adaptation response to neurobehavioral toxicity
UNC-64 and RIC-4, the plasma membrane associated SNAREs syntaxin and SNAP-25, regulate fat storage i
Pre-treatment with mild UV irradiation suppresses reproductive toxicity induced by subsequent cadmiu
Genes required for the functions of olfactory AWA neuron regulate the longevity of Caenorhabditis el
Regulation of longevity by genes required for the functions of AIY interneuron in nematode Caenorhab
Confirmation of the combinational effects of calcium with other metals in a paper recycling mill eff
Pretreatment with mild UV irradiation increases the resistance of nematode Caenorhabditis elegans to
Induction of reproductive deficits in nematode Caenorhabditis elegans exposed to metals at different
Exposure to metals induces morphological and functional alteration of AFD neurons in nematode Caenor
Pre-treatment with mild metal exposure suppresses the neurotoxicity on locomotion behavior induced b
Induction of chemotaxis to sodium chloride and diacetyl and thermotaxis defects by microcystin-LR ex
Prolonged manganses exposure induces severe deficits in lifespan, development and reproduction possi
Adverse effects of metal exposure on chemotaxis towards water-soluble attractants regulated mainly b
Exposure to lead and mercury in young larvae induces more severe deficits in neuronal survival and s
The neurotoxic effects of heavy metal exposure on GABAergic system in nematode Caenorhabditis elegan
The lethality toxicities induced by metal exposure during development in nematode Caenorhabditis ele
HLB-1 functions as a new regulator for the organization and function of neuromuscular junctions in n
Aluminum nanoparticle exposure in L1 larvae results in more severe lethality toxicity than in L4 lar
Close association of intestinal autofluorescence with the formation of severe oxidative damage in in
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