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Foxo 3a regulates apoptosis by negatively targeting mir-21
期刊名称:Journal of Biological Chemistry
时间:0
页码:16958-16966
语言:英文
相关项目:ARC抗心肌肥大的分子机理研究
作者:
王昆,李培峰|
同期刊论文项目
ARC抗心肌肥大的分子机理研究
期刊论文 10
同项目期刊论文
Down-regulation of catalase and oxidative modification of protein kinase CK2 lead to the failure of
Foxo 3a inhibits cardiomyocyte hypertrophy through transactivating catalase.
Apoptosis Repressor with Caspase Recruitment Domain Contributes to Chemotherapy Resistance by Abolis
miR-23a functions downstream of NFATc3 to regulate cardiac hypertrophy
Micrornas coordinate an alternative splicing network during mouse postnatal heart development.
Mir-9 and nfatc3 regulate myocardin in cardiac hypertrophy.
Cardiac hypertrophy is positively regulated by miR-23a
Novel Cardiac Apoptotic Pathway The Dephosphorylation of Apoptosis Repressor With Caspase Recruitmen
p53 initiates apoptosis by transcriptionally targeting the antiapoptotic protein ARC