中文摘要：

氯化对回收的水的安全必要；然而，这个过程关于消毒副产品(DBP ) 和毒性的形成导致担心。这研究在氯化期间在回收的水里为 DBP 和毒性考察了形成和控制策略。调整了，新兴的 DBP 经常在高水平在氯化期间在回收的水里被检测了，显示他们提出到人的更大的风险比那些在里面喝的水。因为 DBP 的形成，尖锐毒性，反雌激素的活动和 cytotoxicity 通常在氯化以后增加了的光细菌和水蚤岩浆。因为有毒的化学药品的破坏，由 umu 测试和雌激素的活动的 Genotoxicity 在氯化以后被减少。在氯化期间，水质量显著地在毒性影响了变化。铵趋于由与氯反应形成氯胺稀释毒性变化，当溴化物趋于由形成 hypobromous 酸加重毒性变化时。在由 ozonation 和凝结的预告的处理期间，消毒副产品形成潜力(DBPFP ) 和毒性形成潜力(TFP ) 偶尔增加移动，它被文档伴随；因此，文档的减少被限制显示 DBPFP 和 TFP 的减少。消除象恐水病的酸和吸水的 neutrals 那样的先锋的关键部分是更重要的。在氯化期间，毒性能与增加的氯剂量和接触时间增加。为了控制过多的毒性形成，相对低的氯剂量和短接触，时间被要求。有效地也与减少的试剂熄灭氯剩余消除了有毒的混合物的形成。

英文摘要：

Chlorination is essential to the safety of reclaimed water; however, this process leads to concern regarding the formation of disinfection byproducts（DBPs） and toxicity. This study reviewed the formation and control strategies for DBPs and toxicity in reclaimed water during chlorination.Both regulated and emerging DBPs have been frequently detected in reclaimed water during chlorination at a higher level than those in drinking water, indicating they pose a greater risk to humans. Luminescent bacteria and Daphnia magna acute toxicity, anti-estrogenic activity and cytotoxicity generally increased after chlorination because of the formation of DBPs. Genotoxicity by umu-test and estrogenic activity were decreased after chlorination because of destruction of toxic chemicals. During chlorination, water quality significantly impacted changes in toxicity.Ammonium tended to attenuate toxicity changes by reacting with chlorine to form chloramine,while bromide tended to aggravate toxicity changes by forming hypobromous acid. During pretreatment by ozonation and coagulation, disinfection byproduct formation potential（DBPFP）and toxicity formation potential（TFP） occasionally increase, which is accompanied by DOC removal; thus, the decrease of DOC was limited to indicate the decrease of DBPFP and TFP. It is more important to eliminate the key fraction of precursors such as hydrophobic acid and hydrophilic neutrals. During chlorination, toxicities can increase with the increasing chlorine dose and contact time. To control the excessive toxicity formation, a relatively low chlorine dose and short contact time were required. Quenching chlorine residual with reductive reagents also effectively abated the formation of toxic compounds.