中文摘要：

南极冰 microalga 能在在南极冰层包含高咸度的隧道或毛孔幸存并且繁荣。在这研究，它被发现绿 microalga Chlamydomonas sp 的那房间膜渗透。从南极海冰的 L4 在由于活跃的氧和激进分子的正式就职与 hypersalinity 对待的房间高。然而，增加了超级氧化物 dismutase (草皮) 清除有害自由激进分子有效地保留房间膜正直。另外，丰满的酸表明了的膜的分析丰满的酸增加了的浸透的丰满的酸和 monounsaturated 和多元不堡和的内容丰满的酸为 14 d 在高盐的处理下面减少了，它有效地减少了膜流动性并且最小化损害到房间膜。词法变化证明 hypersalinity 导致了房间体积和淀粉小粒的消费的增加。因为液泡的 detoxification 的增加，然而，在高盐的应力，完全的不干涉 thylakoids，线粒体和房间原子核下面的电子稠密的存款和草皮活动维持了细胞的基本新陈代谢。蛋白质的全球表示的介绍证明八个蛋白质点消失了， 18 个蛋白质点减少了， 18 个蛋白质点显然在高盐的吃惊以后被提高(P < 0.05 ) 。一新肽(pI 6.90；MW 51 kDa ) 首先在 photosystem II 作为轻反应中心蛋白质 CP43 的处理器被证实，它增加了 Chlamydomonas sp 的光合作用能力。与高咸度对待的 L4。

英文摘要：

Antarctic ice microalga can survive and thrive in channels or pores containing high salinity in Antarctic ice layer. In this study, it was found that cell membrane permeability of green microalga Chlaraydomonas sp. L4 from Antarctic sea ice was high in cells treated with hypersalinity due to the induction of active oxygen and radicals. However, increased super oxide dismutase （SOD） scavenged harmful free radicals effectively to keep cell membrane integrity. Also, the analysis of membrane fatty acids demonstrated the content of saturated fatty acids and monounsaturated fatty acids in- creased and polyunsaturated fatty acids decreased under the high-salt treatment for 14 d, which effectively reduced the membrane fluidity and minimized the injury to cell membrane. The morpho- logical changes showed that hypersalinity induced the increase of cell volume and the consumption of starch granules. However, because of the increase in detoxification of vacuoles, electron-dense deposits and SOD activity under high-salt stress, the complete noninterference thylakoids, mito- chondria and cell nucleus maintained cellular fundamental metabolism. Global-expression profiling of proteins showed eight protein spots disappeared, 18 protein spots decreased and 18 protein spots were enhanced after the high-salt shock obviously （P 〈0.05）. One new peptide （pI 6.90; MW 51 kDa） was primarily confirmed as the processor of light reaction center protein CP43 in photosystem II, which increased photosynthesis ability of Chlamydomonas sp. L4 treated with high salinity.