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6.ROS leads to MnSOD upregulation through ERK2 translocation and p53 activation in selenite-induced
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  • 相关项目：内质网应激凋亡途径在硒诱导白血病细胞凋亡中的作用研究
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内质网应激凋亡途径在硒诱导白血病细胞凋亡中的作用研究
期刊论文 18 会议论文 3
同项目期刊论文
1．通过核质转运调节细胞凋亡的蛋白质
1．P53 transcription-independent activity mediates selenite-induced acute promyelocytic leukemia NB4
2．Selenite induces redox dependent Bax activation and apoptosis in colorectal cancer cells.
3. Sodium selenite induces apoptosis by ROS-mediated endoplasmic reticulum stress and mitochondrial
2．内质网应激与细胞死亡
4. Exposure of human leukemia NB4 cells to increasing concentrations of selenite switches the signal
8.The anticancer effects of sodium selenite and selenomethionine on human colorectal carcinoma cell
10.亚硒酸钠诱导NB4细胞中细胞色素C氧化酶下调机制
2．Selenite induces oxidative stress and apoptosis in colon cancer cells
3．Sodium selenite induced the relocation MnSOD from mitochondria to cytosol during apoptosis
11.Downregulation of Protein Kinase Cα Was Involved in Selenite-Induced Apoptosis of NB4 Cells
7. Sodium selenite induces apoptosis in acute promyelocytic leukemia-derived NB4 cells through mito
5. The subcellular distribution of MnSOD alters during sodium selenite-induced apoptosis.
9.Autophagy inhibition through PI3k/AKT increases apoptosis by sodium selenite in NB4 cells
3.内质网应激介导的细胞凋亡
内质网应激介导的细胞凋亡
Ⅲ型磷脂酰肌醇3-激酶复合物与自噬的研究进展