青枯雷尔氏菌无致病力突变株RS-F523接种后能显著提高番茄对青枯病的抗性,但RS-F523在体外对强致病力菌株RS-F052没有直接的抑制作用.RS-F523接种后植株中与系统获得性抗性(systemic acquired resistance,SAR)相关的过氧化物酶(POD)活性、过氧化氢酶(CAT)活性,H2O2含量以及植株可溶性总蛋白都发生了波动变化,其变化方式和特点与RS-F052接种植株后的变化完全不同,提示RS-F523的生防机制可能与其接种后导致植物的SAK有关.
Tomato resistance ability to Ralstonia solanacearum RS-F052 improves after being inoculated with avirulent strain RS-F523, although RS-F523 cannot inhibit the growing of RS-F052 in vitro. The activities of POD and CAT,the amount of H2O2 and the soluble total proteins,which are all believed to associate with systemic acquired resistance,are tested in the plant which is inocuLated with RS-F523. The results show that each of the indexes above fluctuates and differs from that of the plant inoculated with RS-F052, indicating that the biocontrol mechanism of RS-F523 is associated with SAR.