瑞斯植物建筑学是影响谷物收益的一个重要农学的特点。为了理解控制的分子的机制,种建筑学,一直到有黑暗绿色的叶子的 ering 矮子异种源于与他们对待的 indica 栽培变种 IR64 被描绘。异种,指定了(t) 为 tddl,对非突变而产生之遗传知道到 ering 矮子异种为止。它被一后退的原子基因控制, TDDL (T) ,并且根据 Takeda 的定义组织了进 dn 类型侏儒症。异种的侏儒症基于二个调停 GA 的过程的分析独立于 gibberellic 酸。和在脉管的捆的实质房间的减少的尺寸, brassinosteroid (BR ) 和 tddl (t) 异种的 naphthal-3-acetic 酸(NAA ) 的独立显示 TDDL (T) 基因可能参予另一条荷尔蒙小径。TDDL (T) 是在米饭染色体 4 的长手臂上在 85.51 kb 区域以内印射的罚款,在 20 ORF 被 RiceGAAS 预言的地方(http://ricegaas.dna.affrc。go.jp/rgadb/) 。TDDL (T) 的进一步的克隆将两个都有益于标记植物建筑学的帮助选择(妈) 和在米饭位于下面到 ering 矮子为止的分子的机制的解剖。
Rice plant architecture is an important agronomic trait that affects the grain yield. To understand the molecular mechanism that controls plant architecture, a tillering dwarf mutant with darker-green leaves derived from an indica cultivar IR64 treated with EMS is characterized. The mutant, designated as tddl(t), is nonallelic to the known tiilering dwarf mutants. It is controlled by one recessive nuclear gene, TDDL(T), and grouped into the dn-type dwarfism according to Takeda's definition. The dwarfism of the mutant is independent of gibberellic acid based on the analyses of two GA-mediated processes. The independence of brassinosteroid (BR) and naphthal-3-acetic acid (NAA) of the tddl(t) mutant, together with the decreased size of parenchyma cells in the vascular bundle, indicates that the TDDL(7) gene might participate in another hormone pathway. TDDL(T) is fine mapped within an 85.51 kb region on the long arm of rice chromosome 4, where 20 ORFs are predicted by RiceGAAS (http://ricegaas.dna.affrc. go.jp/rgadb/). Further cloning of TDDL(T) will benefit both marker assisted selection (MAS) of plant architecture and dissection of the molecular mechanism underlying tillering dwarf in rice.