中文摘要：

为了解外来物种红耳龟在不同盐度水域中的生存状况，本研究选用体重67．28g±19．39g的红耳龟进行为期70d的不同盐度胁迫实验，分别测定红耳龟在对照组以及盐度为10％o组和20％e组（以下简写为10组和20组）的体重特定增长率和血液生化指标变化。结果表明，盐度10组的体重特定增长率极显著高于对照组和盐度20组（P〈0．01），而盐度20组的体重特定增长率略大于对照组，但差异不显著（P〉0．05）；盐度10组的肌酸激酶（CK）、谷草转氨酶（AST）、乳酸脱氢酶（LDH）、碱性磷酸酶（ALP）的活性显著高于对照组（P〈0．05）；盐度10组和20组的血糖（Glu）含量显著高于对照组（P〈0．05）；各盐度组血清渗透压（Osmp）、Na＋、C1-、K＋、Mg2＋、血清尿素氮（BUN）、尿酸（UA）含量差异显著（P〈0．05）；盐度20组Ca。’显著高于对照组（P〈0．05）。说明红耳龟可通过提高血液中血糖含量及代谢所需的酶活性使得其代谢水平升高，从而为抵抗胁迫提供所需能量；还可以通过提高血液渗透压及无机离子的浓度来适应外界渗透压的升高，从而使其能够在不同盐度水域中生存。本研究为红耳龟对盐度的耐受生理及入侵机理研究提供生理学方面的依据。

英文摘要：

In order to study the survival mechanisms of exotic Trachemys scripta elegans （TSE） in different ambient salini- ty, a 70-day experiment consist of control, salinity 10%o and 20%0 groups （ the below abbreviations for 10 or 20） was carried on this species （average weight 67.28 g ± 19.39 g） to examine the changes of specific growth rate and blood biochemical ingredient. The results showed that the specific growth rate of salinity l0 group is highly-significandy higher than those of control and salinity 20 group （ P 〈 0.01 ）, respectively. The activities of creatine kinase （ CK）, aspertate aminotransferase （AST）, lactate dehydrogenase （LDH）, alkaline phosphatase （ALP） in serum in salinity 10 group is significantly higher than those of control group （P 〈0.05 ）. The levels of serum glucose （Glu） in salinity 10 and 20 groups are significantly higher than that of control group （ P 〈 0.05 ）. The serum osmotic pressure （ Osmp）, Na＋, C1 -, K＋ , Mg22＋ , serum urea nitrogen （BUN）, uric acid （UA） levels are significantly distinct between different groups （P 〈 0.05 ）. The level of serum Ca2 ＋ in salinity 20 group is significantly higher than that of control group （P 〈 0.05 ）. Therefore, we conclude that T. s. elegans increased serum glucose level; the activity of metabolized enzymes and the serum osmotic pressure by accumulation of ions, urea nitrogen and uric acid in serum when being subjected to ambient salinity stress, which provided the theoretical basis of salinity tolerance and physiological mechanism for invasion.