中文摘要：

本文以小麦（Triticum aestivum L.）品种“豫麦49”幼苗为材料,利用CaCl2与NO供体硝普钠（sodium nitroprusside,SNP）处理盐胁迫下小麦幼苗,研究Ca^2＋和NO缓解NaCl胁迫对小麦生长抑制的机理.结果显示：NaCl胁迫下,外施10mmol·L^-1CaCl2可很好地抑制小麦茎叶电导率升高及根中丙二醛（malonaldehyde,MDA）含量上升,并能促进小麦叶片中叶绿素及根部脯氨酸（proline,Pro）的积累,从而缓解盐伤害;50μmol·L^-1SNP处理可获得与CaCl2处理相似的结果.CaCl2与SNP共同处理可明显增强盐胁迫下小麦幼苗根部Pro积累,提高小麦幼苗过氧化物酶（peroxidase,POD）活性等缓解盐胁迫,但50μmol·L^-1SNP与1mmol·L^-1乙二醇双（2-氨基乙醚）四乙酸[ethyleneglycol bis（2-aminoethyl ether）tetraacetic acid,EGTA]同时处理则部分地抑制SNP对盐害的缓解作用.推测,NO可能主要通过激活小麦根部细胞质膜Ca^2＋通道促进Ca^2＋的吸收,改变胞内Ca^2＋浓度来发挥其对NaCl胁迫伤害的缓解作用.

英文摘要：

To investigate the mechanism of exogenous Ca^2＋ and NO alleviting salt stress in wheat seedlings （ Triticum aestivum L. ）, this paper treated wheat seedlings Yumai 49 with CaCl2 or SNP in response to salt stress. The results showed that 10 mmol ·L^-1 CaCl2 had a better effect on droping the membrane permeability, promoting the chlorophyll accumulation, raising proline accumulation and the MDA degradation of root or leaves to alleviate salt injury. Meanwhile, 50 μmol · L^-1 SNP had similar effect with CaCl2 on droping the membrane permeability, promoting the chlorophyll accumulation, raising proline accumulation and the MDA degradation of root or leaves. SNP＋ CaCl2 significantly increased proline accumulation at root of wheat seedlings and enhance POD activity. However, SNP＋ 1 mmol · L^-1 EGTA inhibited the effects of NO on alleviating salt injury. These results suggest that exogenous NO enhance wheat sah resistance and alleviate salt injury possible by enhancing Ca^2＋ influx by activating root Ca^2＋ channels and improving concentration of Ca^2＋ of wheat seedlings.