运动性骨胳肌微损伤多见于剧烈的耐力和力量训练(以离心练习为主)中。根据骨骼肌的超微结构,可以认为损伤后骨骼肌的组织和形态学变化主要发生在肌浆膜、收缩蛋白和结构蛋白等三方面。骨骼肌微损伤的同时也伴随着力的损失,而力的损失归结于以下几方面的原因:兴奋-收缩偶联功能的缺失,产生收缩力的结构单位比如肌动蛋白(actin)和肌球蛋白(myosin)的瓦解或缺失,力的传送结构的瓦解或缺失。通过大鼠急性运动损伤后对骨骼肌收缩力和拉断力进行测试,对损伤后肌肉收缩特性和材料力学特性进行研究;通过选取对骨骼肌肌力有重要影响的肌球蛋白,研究急性离心运动后骨骼肌损伤后收缩蛋白含量的变化和骨骼肌收缩力和拉断力的关系。
The purpose of this study is to investigate the change of the maximum contractile force, breaking load force and the contents of cytoskeleton protein of the gastrocnemins affected by eccentric exercise. The paper analyzes the relationship between the maximum contractile force and breaking load force of the gastrocnemins and the loss of the cytoskeleton protein and the change of the serum enzyme after the exercise induced the muscle injury. It also discusses the function of cytoskeleton protein in skeleton muscle fiber.