位置：成果数据库 > 期刊 > 期刊详情页
The bad guy cooperates with good cop p53: Bad is transcriptionally up-regulated by p53 and forms a b
  • ISSN号：0270-7306
  • 期刊名称：Molecular and Cellular Biology
  • 时间：0
  • 页码：9071-9082
  • 语言：英文
  • 相关项目：细胞核在细胞凋亡过程中的信号转导及其分子机制的研究
作者： Heese, Klaus|Wu, Mian|Du, Wenjing|Jiang, Peng|
同期刊论文项目
细胞核在细胞凋亡过程中的信号转导及其分子机制的研究
期刊论文 24 会议论文 1 获奖 8
同项目期刊论文
E2F1 represses beta-catenin/TCF activity by direct upregulation of Siah1.
Suppression of tumor growth using a recombinant adenoviral vector carrying the dominant-negative mut
Novel link between E2F1 and Smac/DIABLO: proapoptotic Smac/DIABLO is transcriptionally upregulated b
Livin promotes Smac/DIABLO degradation by ubiquitin-proteasome pathway
Truncated RIP3 (tRIP3) acts upstream of FADD to induce apoptosis in the human hepatocellular carcino
B23 acts as a nucleolar stress sensor and promotes cell survival via its dynamic interaction with hn
Multiple roles for nuclear localization signal (NLS, aa 442-472) of receptor interacting protein 3 (
Tumor suppressor ARF promotes non-classic proteasome-independent polyubiquitination of COMMD1
The Pirh2-keratin 8/18 interaction modulates the cellular distribution of mitochondria and UV-induce
ARF antagonizes the ability of Miz-1 to inhibit p53-mediated transactivation
Suppression of p53 activity by Siva1
Cleavage of RIP3 inactivates its caspase-independent apoptosis pathway by removal of kinase domain
p45, an ATPase subunit of the 19S proteasome, targets the polyglutamine disease protein ataxin-3 to
Noxa/Mcl-1 balance regulates susceptibility of cells to camptothecin-induced apoptosis
p53 and Bad: remote strangers become close friends
Phosphorylation of Pirh2 by Calmodulin-dependent kinase II impairs its ability to ubiquitinate p53
Death effector domain DEDa, a self-cleaved product of caspase-8/Mch5, translocates to the nucleus by
Siah-1S, a novel splice variant of Siah-1 (seven in absentia homolog), counteracts Siah-1-mediated d
细胞命运,生死攸关