中文摘要：

我们的以前的工作证明 Sinorhizobium meliloti 的 enoyl-ACP reductase 基因 fabI1 在 nifA 异种小瘤细菌是下面调整的。获得 fabI1 基因的更好的理解，单个地点插入异种在这研究被构造。fabI1 异种在与 NaCl 的高集中在媒介上成长的细胞生长，和它的能力被延迟被减少。另外，异种在聚集的显型是完全有缺点的。在共生期间， fabI1 异种在主人工厂上推迟了小瘤形成。尽管 FabI1 蛋白质在 S 与另一 enoyl-ACP reductase FabI2 显示出 66% 身份。meliloti，在 fabI1 的缺点没被 fabI2 的 plasmidborne 版本救。这在 S 显示了二 FabI 蛋白质的不同功能。meliloti。

英文摘要：

Our previous work showed that an enoyl-ACP reductase gene fabI1 of Sinorhizobium meliloti was down-regulated in the nifA mutant nodule bacteria. To gain a better understanding offabI1 gene, a single site insertion mutant was constructed in this study. The fabI1 mutant was retarded in cell growth, and its ability to grow on media with high concentration of NaCl was reduced. In addition, the mutant was completely defective in swarming phenotype. During symbiosis, thefabI1 mutant had delayed nodule formation on host plants. Despite the fact that FabI1 protein showed 66% identity with another enoyl-ACP reductase FabI2 in S. meliloti, defects infabI1 were not rescued by the plasmidborne version offabI2. This indicated the different functions of the two FabI proteins in S. meliloti.