位置：成果数据库 > 期刊 > 期刊详情页
细菌脂多糖发育毒作用研究进展
  • 期刊名称：国外医学(卫生学分册)
  • 时间：0
  • 页码：120-123
  • 语言：中文
  • 分类：R965.1[医药卫生—药理学;医药卫生—药学] R994.1[医药卫生—毒理学;医药卫生—药学]
  • 作者机构：[1]安徽医科大学卫生毒理学系,安徽合肥230032
  • 相关基金：国家自然科学基金（30671786）
  • 相关项目：Toll样受体信号通路下游分子ROS、PGs、NO和TNF-α在LPS致畸中的作用
作者： 王华|徐德祥|
关键词： 细菌脂多糖, 发育毒性, 细菌感染
中文摘要：

细菌脂多糖现已被广泛地用于建立炎症或细菌感染的动物模型，而且细菌脂多糖的发育毒性作用研究也得到国内外学者的普遍关注。本文就近年来细菌脂多糖引起胚胎／胎儿死亡、早产、胎儿生长发育迟缓、致畸作用和子代神经发育损伤及其作用机制作一综述。

同期刊论文项目
Toll样受体信号通路下游分子ROS、PGs、NO和TNF-α在LPS致畸中的作用
期刊论文 20
同项目期刊论文
Effect of N-acetylcysteine on lipopolysaccharide-induced intra-uterine fetal death and intra-uterine
Melatonin protects against lipopolysaccharide-induced intra-uterine fetal death and growth retardati
Reactive oxygen species are involved in lipopolysaccharide-induced intrauterine growth restriction a
Tumor necrosis factor alpha partially contributes to lipopolysaccharide-induced intra-uterine fetal
Ascorbic acid protects against lipopolysaccharide-induced intra-uterine fetal death and intra-uterin
Tumor necrosis factor alpha partially contributes to lipopolysaccharide-induced downregulation of CY
Maternally administered lipopolysaccharide (LPS) upregulates the expression of heme oxygenase-1 in f
Lipopolysaccharide (LPS) up-regulates the expression of haem oxygenase-1 in mouse placenta
Maternally administered melatonin differentially regulates lipopolysaccharide-induced proinflammator
Effects of low-dose lipopolysaccharide (LPS) pretreatment on LPS-induced intra-uterine fetal death a
活性氧和TNF-α在LPS上调小鼠胎盘血红素氧化酶-1表达中的作用
Reactive oxygen species contribute to lipopolysaccharide-induced teratogenesis in mice
Differential effects of pyrrolidine dithiocarbamate on TNF-alpha-mediated liver injury in two differ
Maternally-administered lipopolysaccharide (LPS) increases tumor necrosis factor alpha in fetal live
褪黑素对GalN/LPS引起小鼠急性肝损伤的保护作用
活性氧在细菌脂多糖上调小鼠胎肝血红素氧化酶-1表达中的作用
N-乙酰半胱氨酸对GalN/LPS引起小鼠急性肝损伤的保护作用