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A NEW CELLULAR MODEL OF PATHOLOGICAL TDP-43: THE NEUROTOXICITY OF STABLY EXPRESSED CTF25 OF TDP-43 D
期刊名称:neuroscience
时间:2014
页码:88-98
相关项目:双甲氧姜黄素治疗ALS转基因小鼠的前景及机制研究
作者:
李春岩|
同期刊论文项目
双甲氧姜黄素治疗ALS转基因小鼠的前景及机制研究
期刊论文 13
同项目期刊论文
Mitochondrial dysfunction in human TDP-43 transfected NSC34 cell lines and the protective effect of
HO-1 induction in motor cortex and intestinal dysfunction in TDP-43 A315T transgenic mice
Neuroprotection by Monocarbonyl Dimethoxycurcumin C: Ameliorating the Toxicity of Mutant TDP-43 via
Oxidative Stress and Autophagic Alteration in Brainstem of SOD1-G93A Mouse Model of ALS
CURCUMIN ABOLISHES MUTANT TDP-43 INDUCED EXCITABILITY IN A MOTONEURON-LIKE CELLULAR MODEL OF ALS
Autophagy-linked FYVE protein (Alfy) promotes autophagic removal of misfolded proteins involved in a
Food restriction-induced autophagy modulates degradation of mutant SOD1 in an amyotrophic lateral sc
The modest impact of transcription factor Nrf2 on the course of disease in an ALS animal model.
Full-length TDP-43 and its C-terminal fragments activate mitophagy in NSC34 cell line
TERT基因转染BMSC血管性痴呆大鼠记忆功能及海马CA1区突触可塑性的影响