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Death domain-associated protein 6 (Daxx) selectively represses IL-6 transcription through histone de
ISSN号:0021-9258
期刊名称:J Biol Chem
时间:2014.3.28
页码:9372-9379
相关项目:天然免疫识别与调控的新型分子机制研究
作者:
王春梅|姜明红|李楠|曹雪涛|
同期刊论文项目
天然免疫识别与调控的新型分子机制研究
期刊论文 27
会议论文 13
获奖 6
同项目期刊论文
Regulation of Toll-like receptor signaling pathways in innate immune responses
Induction of Siglec-G by RNA viruses inhibits the innate immune response by promoting RIG-I degradat
LRRFIP2 negatively regulates NLRP3 inflammasome activation in macrophages by promoting Flightless-I-
Histone methyltransferase Ash1l suppresses interleukin-6 production and inflammatory autoimmune dise
Immune responsive gene 1 (IRG1) promotes endotoxin tolerance by increasing A20 expression in macroph
Inductionof Siglec-G by RNA viruses inhibits the innate immune response by promoting RIG-Idegradatio
Type I IFN inhibits innateIL-10 production in macrophages through histone deacetylase 11 bydownregul
Self-regulationand cross-regulation of pattern-recognition receptor signalling in health anddisease.
Regulationof Toll-like receptor signaling pathways in innate immune responses.
IFN-γprimes macrophage activation by increasing phosphatase and tensin homolog viadownregulation of
Rhbdd3 controls autoimmunity by suppressing the production of IL-6 by dendritic cells via K27-linked
The STAT3-binding long noncoding RNA lnc-DC controls human dendritic cell differentiation.
IFN-γ Primes Macrophage Activation by Increasing Phosphatase and Tensin Homolog via Downregulation o
Tet2 is required to resolveinflammation by recruiting Hdac2 to specifically repress IL-6
Innatesignaling in the inflammatory immune disorders.
Siglec1 suppressesantiviral innate immune response by inducing TBK1 degradation via the ubiquitinlig
TheSTAT3-binding long noncoding RNA lnc-DC controls human dendritic celldifferentiation
Histone lysinemethyltransferase Ezh1 promotes TLR-triggered inflammatory cytokine productionby suppr
Fine-tuning MAVS- and STING-mediated antiviral innate immunity
Cytoplasmic STAT4 Promotes Antiviral Type I IFNProduction by Blocking CHIP-Mediated Degradation of R
Type I IFN-Inducible Downregulation of MicroRNA-27aFeedback Inhibits Antiviral Innate Response by Up
Notch signal suppresses Toll-like receptor-triggered inflammatory responses in macrophages by inhibi