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Loss of glutathione S ‐ transferase A4 accelerates obstruction ‐ induced tubule damage and renal fib
期刊名称:The Journal of ...
时间:2012
页码:448-458
相关项目:炎症与细胞内外环境调控EMT的机制
作者:
...|MH Wilson|R Sharma|YC Awasthi|J Du...|
同期刊论文项目
炎症与细胞内外环境调控EMT的机制
期刊论文 30
同项目期刊论文
Atg5 deficiency-mediated mitophagy aggravates cardiac inflammation and injury in response to angiote
自噬相关基因5下调抑制血管紧张素Ⅱ介导的巨噬细胞凋亡
Akt2 mediates TGF-beta1-induced epithelial to mesenchymal transition by deactivating GSK3beta/s
Loss of glutathione S -transferase A4 accelerates obstruction-induced tubule damage and renal fibros
Kruppel-like factor 4 transcriptionally regulates TGF-beta1 and contributes to cardiac myofibroblast
Knockout of CD8 delays reendothelialization and accelerates neointima formation in injured arte
Reciprocal interaction between macrophages and T cells stimulates IFN -γ and MCP - 1 produ
Adiponectin suppresses angiotensin II-induced inflammation and cardiac fibrosis through activat
FSP-1 Silencing in Bone Marrow Cells Suppresses Neointima Formation in Vein Graft
Chemokine receptor CX3CR1 contributes to macrophage survival in tumor metastasis
Macrophage-stimulated cardiac fibroblast production of IL-6 is essential for TGF beta/Smad act
TNF-alpha and IL-1beta neutralization ameliorates angiotensin II-induced cardiac damage in male
The Notch γ-secretase inhibitor ameliorates kidney fibrosis via inhibition of TGF-β/Smad2/3 signalin
高血压对小鼠血管骨桥蛋白表达的影响
Loss of glutathione S-transferase A4 accelerates obstruction-induced tubule damage and renal fi
Akt2 is involved in loss of epithelial cells and renal fibrosis following unilateral ureteral o
Complement 5a receptor mediates angiotensin II-induced cardiac inflammation and remodeling.
Aging increases CCN1 expression leading to muscle senescence.
Senescent Cardiac Fibroblast Is Critical for Cardiac Fibrosis after Myocardial Infarction
Interleukin - 6 / signal transducer and activator of transcription 3 ( STAT3 ) pathway is e