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PPARγ通过调节脂质代谢和VSMC增殖参与动脉粥样硬化的发生与发展
  • 所属机构名称：河北医科大学
  • 会议名称：2012年第十一届全国脂质与脂蛋白学术会议
  • 成果类型：会议
  • 相关项目：KLF4对血管平滑肌细胞发挥促增殖与促分化双重作用的分子机制
作者： 温进坤|温进坤|张新华|张新华|郑 斌|郑 斌|
同会议论文项目
KLF4对血管平滑肌细胞发挥促增殖与促分化双重作用的分子机制
期刊论文 16 会议论文 12
同项目会议论文
Retinoic acid receptor alpha mediates Klf4 gene expression induced by all-trans retinoic acid in vas
Retinoic acid receptor alpha mediates Klf4 gene expression induced by all-trans retinoic acid in vas
TGF-β1 regulates AT1 receptor expression via PKC-δ- and Smad-mediated signal pathways
TGF-β1 regulates AT1 receptor expression via PKC-δ- and Smad-mediated signal pathways
RARalpha mediates all-trans retinoic acid-induced KLF4 gene expression through interacting with KLF4
PPARγ通过调节脂质代谢和VSMC增殖参与动脉粥样硬化的发生与发展
Functional interaction of KLF5 with Sp1 upregulates apelin in vascular smooth muscle cells
Functional interaction of KLF5 with Sp1 upregulates apelin in vascular smooth muscle cells
TGF-β1 downregulates AT1 receptor expression via PKC-δ-mediated Sp1 dissociation from KLF
TGF-β1 downregulates AT1 receptor expression via PKC-δ-mediated Sp1 dissociation from KLF
RARalpha mediates all-trans retinoic acid-induced KLF4 gene expression through interacting with KLF4