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TGF-β1 downregulates AT1 receptor expression via PKC-δ-mediated Sp1 dissociation from KLF
  • 所属机构名称：河北医科大学
  • 会议名称：2012年全国生物化学与分子生物学学术大会
  • 成果类型：会议
  • 相关项目：KLF4对血管平滑肌细胞发挥促增殖与促分化双重作用的分子机制
作者： Xin-hua Zhang|Xin-hua Zhang|Bin Zheng|Bin Zheng|Chun Gu|Chun Gu|Jian-ran Fu|Jian-ran Fu|Jin-kun Wen|Jin-kun Wen|
同会议论文项目
KLF4对血管平滑肌细胞发挥促增殖与促分化双重作用的分子机制
期刊论文 16 会议论文 12
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Retinoic acid receptor alpha mediates Klf4 gene expression induced by all-trans retinoic acid in vas
Retinoic acid receptor alpha mediates Klf4 gene expression induced by all-trans retinoic acid in vas
TGF-β1 regulates AT1 receptor expression via PKC-δ- and Smad-mediated signal pathways
TGF-β1 regulates AT1 receptor expression via PKC-δ- and Smad-mediated signal pathways
RARalpha mediates all-trans retinoic acid-induced KLF4 gene expression through interacting with KLF4
PPARγ通过调节脂质代谢和VSMC增殖参与动脉粥样硬化的发生与发展
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Functional interaction of KLF5 with Sp1 upregulates apelin in vascular smooth muscle cells
Functional interaction of KLF5 with Sp1 upregulates apelin in vascular smooth muscle cells
TGF-β1 downregulates AT1 receptor expression via PKC-δ-mediated Sp1 dissociation from KLF
RARalpha mediates all-trans retinoic acid-induced KLF4 gene expression through interacting with KLF4