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Ferroportin1 和 hephaestin 在 MES23.5 细胞铁转出过程中的作用。
  • 所属机构名称：青岛大学
  • 会议名称：中国生理学会第22届会员代表大会暨庆祝中国生理学会成立80周年学术大会
  • 成果类型：会议
  • 会场：北京
  • 相关项目：二价金属离子对多巴胺能神经元损伤机制的深入探讨
作者： 姜宏|王俊|宋宁|谢俊霞|
同会议论文项目
二价金属离子对多巴胺能神经元损伤机制的深入探讨
期刊论文 8 会议论文 17
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铜离子引起大鼠黑质多巴胺能神经元的退行性变(英文)
Overexpression of Divalent metal transporter 1 enhanced iron induced cell damage.
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Iron regulatory proteins were involved in the 6-hydroxydopamine (6-OHDA) induced ferroportin1 down-r
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Peripheral zinc (Zn2+) induced dopaminergic neurons apoptosis in the nigrostriatal system of rats.
Potassium channels were involved in zinc-induced apoptosis in MES23.5 cells.
Function of increased human divalent metal transporter 1 in iron accumulation.
The role of iron export proteins in the nigral iron accumulation of 6-OHDA lesioned rat.
Over-expressed ferroportin1 and/or hephaestin attenuated iron induced oxidative stress in MES23.5 ce
Neurotoxic effect of zinc on MES23.5 dopaminergic cells.