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6-OHDA单侧损毁大鼠黑质铁转出蛋白的表达降低
  • 所属机构名称：青岛大学
  • 会议名称：中国神经科学学会第六届学术会议暨学会成立十周年庆祝大会
  • 成果类型：会议
  • 会场：北京
  • 相关项目：二价金属离子对多巴胺能神经元损伤机制的深入探讨
作者： 王俊|姜宏|谢俊霞|
同会议论文项目
二价金属离子对多巴胺能神经元损伤机制的深入探讨
期刊论文 8 会议论文 17
同项目会议论文
Ferroportin1 和 hephaestin 在 MES23.5 细胞铁转出过程中的作用。
Ceruloplasmin is Involved in the Nigral Iron Accumulation of 6-OHDA Lesioned Rats.
Neuroprotective effects of iron chelator desferal on dopaminergic neurons in the substantia nigra of
Up-regulated divalent metal transporter 1 by 1-Methy-4-phenylpyridinium iodide (MPP+) enhances iron
铜离子引起大鼠黑质多巴胺能神经元的退行性变(英文)
Overexpression of Divalent metal transporter 1 enhanced iron induced cell damage.
Rg1 protects dopaminergic neurons in the substantia nigra of MPTP-treated C57BL6 mice by its iron-ch
Peripheral zinc (Zn2+) induced dopaminergic neurons apoptosis in the nigrostriatal system of rats.
Iron regulatory proteins were involved in the 6-hydroxydopamine (6-OHDA) induced ferroportin1 down-r
Potassium channels are involved in zinc-induced apoptosis in MES23.5 cells
Peripheral zinc (Zn2+) induced dopaminergic neurons apoptosis in the nigrostriatal system of rats.
Potassium channels were involved in zinc-induced apoptosis in MES23.5 cells.
Function of increased human divalent metal transporter 1 in iron accumulation.
The role of iron export proteins in the nigral iron accumulation of 6-OHDA lesioned rat.
Over-expressed ferroportin1 and/or hephaestin attenuated iron induced oxidative stress in MES23.5 ce
Neurotoxic effect of zinc on MES23.5 dopaminergic cells.