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O-GlcNAcylation negtively regulates phosphorylation of tau: a novel mechanism involved in Alzheimer'
  • 所属机构名称：南通大学
  • 会议名称：2008年全国糖生物学会议, 2008/6/8.
  • 成果类型：会议
  • 相关项目：Mnb/Dyrk1A 对tau外显子10可变剪接的调控研究
同会议论文项目
Mnb/Dyrk1A 对tau外显子10可变剪接的调控研究
期刊论文 9 会议论文 24
同项目会议论文
TRUNCATION OF GSK-3b BYACTIVATED CALPAIN I IN ALZHEIMER BRAIN ENHANCES ITS BIOLOGICAL ACTIVITY
Truncation and activation of Dyrk1A in Alzheimer’s disease brain by activated calpain I
P2-147: Modulation of SRp55-regulated alternative splicing oftau exon 10 by Dyrk1A
Dyrk1A modulates SRp55-regulated alternative splicing of tau exon 10
REGULATION OF AKT BY O-GLCNACYLATION IN VARIOUS CELL TYPES: IMPLICATION IN ALZHEIMER’S DISEASE
Tau代谢异常在tau病理疾病(tauopathy)致病过程中的作用
OVEREXPRESSION OF DYRK1A DYSREGULATES THE ALTERNATIVE SPLICING OF TAU EXON 10
Truncation of CREB by calpain I reduces its ability in the promotion of GLUT3 expression
Calpain I对CREB的截断对GLUT3的表达调节的影响影响
蛋白激酶A对tau外显子10可变剪接的调节
Dyrk1A的截断和活化在阿尔茨海默病脑中致病性tau形成中的作用
Truncation of CREB by calpain I reduces its ability in the promotion of GLUT3 expression
S1-03-05: Increased dosage of Dyrk1A alters the alternative splicing of Tau exon 10 in Down Syndrome
P2-145: Regulation of ASF- mediated alternative splicing of tau exon 10 by PKA
P2-148: Involvement of 9G8 in alternative splicing of tau exon 10
Phosphorylation of SC35 by Dyrk1A inhibits its promotion on tau exon 10 inclsuion
SPLICING FACTOR SC35 REGULATES THE ALTERNATIVE SPLICING OF TAU EXON 10
OVEREXPRESSION OF DYRK1A DYSREGULATES THE ALTERNATIVE SPLICING OF TAU EXON 10
S1-03-05: Increased dosage of Dyrk1A alters the alternative splicing of Tau exon 10 in Down Syndrome
P2-145: Regulation of ASF- mediated alternative splicing of tau exon 10 by PKA
P2-148: Involvement of 9G8 in alternative splicing of tau exon 10
Phosphorylation of SC35 by Dyrk1A inhibits its promotion on tau exon 10 inclsuion
SPLICING FACTOR SC35 REGULATES THE ALTERNATIVE SPLICING OF TAU EXON 10