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<span style="color:black;font-family:Calibri;font-size:16pt;font-weight:bold;">Peak
  • ISSN号：0172-4622
  • 期刊名称：International Journal of Sports Medicine
  • 时间：2011.12.1
  • 页码：970-974
  • 相关项目：FGFR3在小鼠关节软骨损伤修复中的作用及机制研究
作者： Jing Sun|Fengtao Luo|Qidi Sun|Ling Zhao|Nan Su|Xiaolan Du|Haiyang Huang|Yue Shen|Lin Chen|
同期刊论文项目
FGFR3在小鼠关节软骨损伤修复中的作用及机制研究
期刊论文 26 会议论文 23
同项目期刊论文
FGFR3 induces degradation of BMP type I receptor to regulate skeletal development
Intermittent PTH (1-34) injection rescues the retarded skeletal development and postnatal lethality
Inactivation of Vhl in Osteochondral Progenitor Cells Causes High Bone Mass Phenotype and Protects A
间断PTH 处理调节骨稳态的分子机制
1例先天性脊柱骨骺发育不良患者COL2A1基因新突变
Role of FGFs/FGFRs in skeletal growth and bone regeneration
构建FGFR3基因沉默慢病毒载体及其对ATDC5细胞增殖和分化的影响
Role of FGF/FGFR signaling in skeletal development and homeostasis: learning from mouse models
手术诱导的小鼠骨关节炎模型中关节软骨MMP-13表达上调
Role of FGFs/FGFRs in skeletal development and bone regeneration
Overexpression of H1 calponin in osteoblast lineage cells leads to a decrease in bone mass by disrup
阻断TGF-β/TGF-βRⅠ通路对ATDC5细胞增殖、分化及细胞外基质的影响
Serum Bone Alkaline Phosphatase in Assessing Illness Severity of Infected Neonates in the Neonatal I
II型常染色体显性遗传骨硬化症一个家系的临床和遗传学分析
Genetic Inhibition of Fibroblast Growth Factor Receptor 1 in Knee Cartilage Attenuates the Degenerat
Efficient multi-site-directed mutagenesis directly from genomic template.
间断PTH处理调节骨稳态的分子机制
骨培养探讨脂多糖对小鼠骨骼发育及矿化的影响
成年小鼠骨细胞的分离鉴定及其成纤维细胞生长因子受体的表达