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PTEN deficiency causes dyschondroplasia in mice by enhanced hypoxia-inducible factor-1( signaling an
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  • 相关项目：骨骼发育及其相关疾病的分子机理研究
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骨骼发育及其相关疾病的分子机理研究
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microRNA在心脏发育与疾病中功能研究进展
成骨细胞特异性表达Cre重组酶转基因小鼠的建立
SMAD4 is required for maintaining normal murine postnatal bone homeostasis
Smad4 is required for the normal organization of the cartilage growth plate
Accelerated re-epithelialization in Dpr2-deficient mice is associated with enhanced response to TGF-
Transforming growth factor-β regulated miR-24 promotes skeletal muscle differentiation
成骨细胞特异性剔除Smad4不影响小鼠造血稳态的维持
Altered Gene Expression in Articular Chondrocytes of Smad3ex8/ex8 Mice Revealed by Gene Profiling Us
The up-regulation of 14-3-3 proteins in Smad4 deficient epidermis and hair follicles at catagen
Transgenic mice that express Cre recombinase in hypertrophic chondrocytes
肥大软骨细胞特异性表达Cre 重组酶转基因小鼠的建立
CollagenI(1 promoter drives the expression of Cre recombinase in Osteoblasts of transgenic mice
Targeting WW domains linker of HECT-type ubiquitin ligase Smurf1 for activation by CKIP-1
TGF-(超家族在软骨发生、发育和维持中的作用
基因打靶技术：开创遗传学新纪元
利用寡核苷酸芯片分析Smad3基因敲除小鼠关节软骨细胞基因表达谱的改变
Smad4介导转化生长因子-β信号调节骨骼发育和稳态维持的功能
Collagenl α1 promoter drives the expression of Cre recombinase in osteoblasts of transgenic mice
TGF—β超家族在软骨发生、发育和维持中的作用
肥大软骨细胞特异性表达Cre重组酶转基因小鼠的建立