中文摘要：

骆驼毛的织物，充当一个压力信号，在植物抵抗起关键作用浇应力。因为骆驼毛的织物信号生产基于骆驼毛的织物生合成， NCED 的规定，在 ABA 生合成小径的关键酶，通常被认为是控制骆驼毛的织物信号生产的唯一的因素。这里，我们证明在有骆驼毛的织物生合成的 synergisticregulation 的联合的那骆驼毛的织物分解代谢在管理骆驼毛的织物信号生产起一个关键作用。水应力导致了骆驼毛的织物的重要累积，它在分开并且属于的叶子展出了不同模式。骆驼毛的织物分解代谢跟随了指数的腐烂的一个时间的趋势为基本并且压力骆驼毛的织物，并且在基本骆驼毛的织物和压力 ABA.Thus 的分解代谢的一半生活有小差别，骆驼毛的织物分解代谢的绝对的率，即骆驼毛的织物的数量每统一时间发生分解代谢，有增加的骆驼毛的织物累积的增加。从骆驼毛的织物生合成和分解代谢的动态过程，强调骆驼毛的织物累积可以被所有步骤的 synergisticregulation 在 ABA 生合成小径管理，这能被推断。而且，骆驼毛的织物支撑了 NCED3 的激活维持应力的提高的水平应该被要求。这推论被编码的基因主修的进一步的调查结果支持在 ABA 生合成小径的酶例如 NCED3， AAO3 和骆驼毛的织物是都走水路激活应力，并且与骆驼毛的织物累积进行， NCED3 的表情， AAO3 和 ABA3 仍然保持激活。骆驼毛的织物分解代谢和基因表示上的数据联合显示那骆驼毛的织物信号生产被 NCED3 和骆驼毛的织物生合成和分解代谢的 thesynergistic 规定的持续激活控制。

英文摘要：

ABA, acting as a stress signal, plays crucial roles in plant resistance to water stress. Because ABA signal production is based on ABA biosynthesis, the regulation of NCED, a key enzyme in the ABA biosynthesis pathway, is normally thought of as the sole factor controlling ABA signal production. Here we demonstrate that ABA catabolism in combination with a synergistic regulation of ABA biosynthesis plays a crucial role in governing ABA signal production. Water stress induced a significant accumulation of ABA, which exhibited different patterns in detached and attached leaves. ABA catabolism followed a temporal trend of exponential decay for both basic and stress ABA, and there was little difference in the catabolic half-lives of basic ABA and stress ABA. Thus, the absolute rate of ABA catabolism, i.e. the amount of ABA catabolized per unit time, increases with increased ABA accumulation. From the dynamic processes of ABA biosynthesis and catabolism, it can be inferred that stress ABA accumulation may be governed by a synergistic regulation of all the steps in the ABA biosynthesis pathway. Moreover, to maintain an elevated level of stress ABA sustained activation of NCED3 should be required. This inference was supported by further findings that the genes encoding major enzymes in the ABA biosynthesis pathway, e.g. NCED3, AAO3 and ABA3 were all activated by water stress, and with ABA accumulation progressing, the expressions of NCED3, AAO3 and ABA3 remained activated. Data on ABA catabolism and gene expression jointly indicate that ABA signal production is controlled by a sustained activation of NCED3 and the synergistic regulation of ABA biosynthesis and catabolism.