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SOX6 and PDCD4 enhance cardiomyocyte apoptosis through LPS-induced miR-499 inhibition.
ISSN号:1360-8185
期刊名称:Apoptosis
时间:2015.12.10
页码:-
相关项目:胰岛因子1促进胰岛细胞增殖的分子机制研究
作者:
Lu, Qin|Chen, Ping|Ma, Kangtao|Zhou, Chunyan|
同期刊论文项目
胰岛因子1促进胰岛细胞增殖的分子机制研究
期刊论文 22
同项目期刊论文
ISL1在肿瘤中的作用及展望
Autophagy eliminates cytoplasmic β-catenin and NICD to promote the cardiac differentiation of P19CL6
Wnt-promoted Isl1 expression through a novel TCF/LEF1 binding site and H3K9 acetylation in early sta
Interaction of Wnt/beta-catenin and notch signaling in the early stage of cardiac differentiation of
ISL-1 is overexpressed in non-Hodgkin lymphoma and promotes lymphoma cell proliferation by forming a
miR-499 protects cardiomyocytes from H2O2-induced apoptosis via its effects on Pdcd4 and Pacs2
A positive feedback regulation of ISL-1 in DLBCL but not in pancreatic beta-cells
microRNA在心脏发育和心脏疾病中的作用
An abnormally high expression of ISL-1 represents a potential prognostic factor in gastric cancer.
干细胞的异常分化与肿瘤发生
A positive feedback regulation of ISL-1 in DLBCL but not in pancreatic β-cells.
ISL-1 promotes pancreatic islet cell proliferation by forming an ISL-1/Set7/9/PDX-1 complex.
α-硫辛酸对在H9c2细胞在H_2O_2导致的氧化应激损伤中的保护作用
JAK/STAT信号转导通路与淋巴瘤的形成
ISL1在肿瘤发生中的作用