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OCT4 is epigenetically regulated by DNA hypomethylation of promoter and exon in primary gliomas
  • ISSN号：1791-2431 (Electronic)
  • 期刊名称：Oncology Reports
  • 时间：2013.4.9
  • 页码：201-206
  • 相关项目：胶质瘤中IDH1突变的肿瘤生长抑制与放化疗增敏作用及其机制
作者： 施金龙|
同期刊论文项目
胶质瘤中IDH1突变的肿瘤生长抑制与放化疗增敏作用及其机制
期刊论文 17
同项目期刊论文
Detection of serum Alu element hypomethylation for the diagnosis and prognosis of glioma.
星形细胞胶质瘤中IDH1基因突变及DNA甲基化分析
Identifying the genes regulated by IDH1 via gene-chip in glioma cell U87
The assessment of immature microvascular density in brain gliomas with dynamic contrast-enhanced mag
OCT4 is epigenetically regulated by DNA hypomethylation of promoter and exon in primary gliomas.
PAX3 is overexpressed in human glioblastomas and critically regulates the tumorigenicity of glioma c
胶质瘤细胞中异柠檬酸脱氢酶1基因突变对肿瘤细胞的生长抑制及作用机制
Decreasing GSH and increasing ROS in chemosensitivity gliomas with IDH1 mutation.
An IDH1 mutation inhibits growth of glioma cells via GSH depletion and ROS generation.
Clinical significance and prognostic value of PAX3 expression in human glioma.
异柠檬酸脱氢酶1-R132H突变体慢病毒载体的构建与鉴定
血清游离DNA甲基化检测对胶质瘤的意义