欢迎您!
东篱公司
退出
申报数据库
申报指南
立项数据库
成果数据库
期刊论文
会议论文
著 作
专 利
项目获奖数据库
位置:
成果数据库
>
期刊
> 期刊详情页
Cracking the molecular origin of intrinsic tyrosine kinase activity through analysis of pathoge
ISSN号:2211-1247
期刊名称:Cell Reports
时间:2013.7.25
页码:376-384
相关项目:FGF21通过抑制GSK-3β活性对糖尿病心肌病的保护作用研究
作者:
Chen, Huaibin#|Huang, Zhifeng#|Dutta, Kaushik#|Blais, Steven|Neubert A.Thomas|Xiaokun Li|Cowburn, David|Traaseth J. Nathaniel|Mohammadi, Moosa|
同期刊论文项目
FGF21通过抑制GSK-3β活性对糖尿病心肌病的保护作用研究
期刊论文 7
会议论文 1
获奖 3
同项目期刊论文
A solid-phase PEGylation strategy for protein therapeutics using a potent FGF21 analog.
A novel solid-phase site-specific PEGylation enhances the in vitro and in vivo biostabilty of r
Structural mimicry of a-loop tyrosine phosphorylation by a pathogenic FGF receptor 3 mutation.
One-step production of bioactive proteins through simultaneous PEGylation and refolding.
Solid-phase N-terminus PEGylation of recombinant human fibroblast growth factor 2 on heparin-se
Fibroblast growth factor 21 protects the diabetic heart from cell death via extracellular signal-reg