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NE strengthens the immunosuppression induced by AlCl3 through β2-AR/cAMP pathway in cultured rat per
  • ISSN号：0163-4984
  • 期刊名称：Biological Trace Element Research
  • 时间：2015.4.23
  • 页码：234-241
  • 相关项目：铝免疫毒性的神经内分泌调控机制
作者： Limin Ren|Chunyu Xiu|Yanfei Li|Yanzhu Zhu|
同期刊论文项目
铝免疫毒性的神经内分泌调控机制
期刊论文 38 著作 1
同项目期刊论文
Effects of Aluminum Trichloride on Structure and Function of Kidney in Chickens
亚慢性铝暴露对雏鸡大脑组织氨基酸类神经递质的影响
Effects of Subchronic Aluminum Exposure on Liver Function in Rats
长期铝暴露对大鼠肝脏功能的影响
铝中毒解毒剂DHPO的研究进展
Impact of aluminum exposure on the immune system: A mini review
Effects of sub-chronic aluminum chloride on spermatogen
Aluminum trichloride disorders bile acid secretion and induces hepatocyte apoptosis in rats
Effects of aluminum exposure on the allergic responses and humoral immune function in rats
  Effects  of  Sub-chronic Aluminum Chloride  Exposure&
NE strengthens theimmunosuppression induced by AlCl3 through β2-AR/cAMP pathway in cultured ratperit
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Suppressive effects of aluminum trichloride on the T lymphocyte immune function of rats
Effects of Aluminum on Immune Functions of Cultured Splenic T and B Lymphocytes in Rats
Effects of aluminum trichloride on the trace elements and cytokines in the spleen of rats
Suppressive effect of accumulated aluminum trichloride on the hepatic microsomal cytochrome P450 enz
Effects of sub-chronic aluminum chloride exposure on rat ovaries
Effects of sub-chronic aluminum chloride on spermatogenesis and testicular enzymatic activity in mal
AlCl3 Induces Lymphocyte Apoptosis in Rats through the Mitochondria-Caspase Dependent Pathway
Aluminum Chloride Induces Osteoblasts Apoptosis via Disrupting Calcium Homeostasis and Activating Ca
cAMP/PKA signaling pathway induces apoptosis by inhibited NF-κB in aluminum chloride-treated lymphoc
Effects of Norepinephrine on Immune Functions of Cultured Splenic Lymphocytes Exposed to Aluminum Tr
Aluminum Induces Osteoblast Apoptos is Through the Oxidative Stress-Media ted JNK Signaling Pathway
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Aluminum chloride and norepinephrine induced immunotoxicity on splenic lymphocytes by activating β2-