中文摘要：

通过巯基与聚氯乙烯（PVC）氯原子的亲核反应将羧基引入PVC大分子链，再利用环氧脂肪酸甲酯与羧基之间的反应，通过化学键将其连接入PVC大分子链，实现对PVC内增塑．用傅里叶红外（FTIR）、核磁共振（^1H-NMR）、示差扫描量热法（DSC）对改性产物进行了表征．FTIR、^1H—NMR结果表明，环氧脂肪酸甲酯成功共价连接到了PVC大分子链上，接枝率为1．2m01％；DSC测试结果显示，所采用的环氧脂肪酸甲酯改性PVC方法能有效降低PVC的玻璃化转变温度（T0），5．6wt％的增塑剂可将PVC的T1从81．4℃降至37．8℃；增塑剂耐溶剂抽出性与耐挥发性实验结果表明，环氧脂肪酸甲酯在测试时间内无损失，增塑剂迁移得到彻底抑制．

英文摘要：

To suppress the migration of epoxy fat acid methyl ester （EFAME） from plasticized PVC, EFAME, as an internal plasticizer, was covalently bound to poly（vinyl chloride） （PVC） chains via a special synthesis process. The carboxyl-containing PVC （PVC-TSA） was synthesized through nucleophilic substitution reaction between PVC and thiosalicylic acid （TSA） , and then EFAME was added to react with PVC-TSA to gain the internally plasticized PVC （PVC-TSA-EFAME）. Fourier transform infrared spectrometry （FTIR） and hydrogen spectrum nuclear magnetic resonance （1H-NMR） confirmed the structures of PVC-TSA and PVC- TSA-EFAME. The 1H-NMR data were further used to calculate the degree of modification. It was found that a degree of modification of 1.2 mol% was achieved using the method of this work. The glass transition temperature （ T ） determination was realized by means of differential scanning calorimetry （DSC）. The Tss of PVC and PVC-TSA-EFAME were respectively 81.4 ℃ and 37.8 ℃. Compared to PVC, the marked decrease of Ts was attributed to the introduction of EFAME which increased the flexibility of PVC chains. The extraction and fogging experiments showed that almost no extraction or atomization loss was detected in PVC-TSA- EFAME, which suggested that the migration of EFAME from the plasticized PVC was completely suppressed.