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Long non-coding RNA CASC2 supresses malignancy in human gliomas by miR-21.
期刊名称:Cellular Signaling.
时间:0
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相关项目:miR-17-92基因簇调节血肿瘤屏障通透性的作用机制
作者:
Li Z|Li ZQ|Ma J|Xue YX|
同期刊论文项目
miR-17-92基因簇调节血肿瘤屏障通透性的作用机制
期刊论文 64
同项目期刊论文
内皮-单核细胞激活多肽酶Ⅱ增强血肿瘤屏障通透性的机制
Doxycycline-mediated protective effect against focal cerebral ischemia–reperfusion injury
地塞米松预处理对氧糖剥夺条件下血脑屏障通透性的影响.
内皮-单核细胞激活多肽-Ⅱ增强血肿瘤屏障通透性的信号通路.
ROCK介导缓激肽开放SD大鼠血肿瘤屏障.
内皮-单核细胞激活多肽对人U118胶质瘤细胞自噬的影响及机制
骨髓间充质干细胞向胶质瘤迁移中血小板衍生生长因子BB的作用
MiR-181a regulates blood-tumor barrier permeability by targeting Krüppel-like factor 6.
miR-101 Acts as a Tumor Suppressor by Targeting Kruppel-like Factor 6 in Glioblastoma Stem Cells.
MiR-152 functions as a tumor suppressor in glioblastoma stem cells by targeting Krüppel-like factor
MiR-18a increased the permeability of BTB via RUNX1 mediated down-regulation of ZO-1, occluding and
Effects of erythropoietin on blood-brain barrier tight junctions in ischemia-reperfusion rats.
Blood-brain barrier permeability change and regulation mechanism after subarachnoid hemorrhage.
Prognostic significance of osteopontin and carbonic anhydrase 9 in human brain tumors: A Meta-Analys
Gas5 Exerts Tumor-suppressive Functions in Human Glioma Cells by Targeting miR-222.
Long non-coding RNA CASC2 suppresses malignancy in human gliomas by miR-21
miR-101 acts as a tumor suppressor by targeting Kruppel-like factor 6 in glioblastoma stem cells
MiR-449a exerts tumor-suppressive functions in human glioblastoma by targeting Myc-associated zinc-f
内皮-单核细胞激活多肽-Ⅱ增强血肿瘤屏障通透性的信号通路
CRM197 in combination with shRNA interference of VCAM-1 displays enhanced inhibitory effects on huma
Effects of insulin combined with idebenone on blood-brain barrier permeability in diabetic rats.
Autophagy Induction by Endothelial-Monocyte Activating Polypeptide II Contributes to the Inhibition
MiR-18a increased the permeability of BTB via RUNX1 mediated down-regulation of ZO-1, occludin and c
大鼠原代脑微血管内皮细胞体外分离与培养的实验研究
蛋白激酶C在内皮-单核细胞激活多肽Ⅱ增强血肿瘤屏障通透性中的作用
过表达miR-34a增加血肿瘤屏障通透性机制的研究
MiR-429对人U87胶质瘤细胞增殖、迁移和侵袭能力的影响及其作用机制
EMAP-Ⅱ上调miR-429表达抑制人U87胶质瘤细胞的增殖、迁移和侵袭能力
地塞米松预处理对氧糖剥夺大鼠血脑屏障通透性的影响
内皮—单核细胞激活多肽酶Ⅱ增强血肿瘤屏障通透性的作用位点
RhoA在内皮-单核细胞激活多肽-II增强血肿瘤屏障通透性中的作用
蒿甲醚对人胶质瘤细胞侵袭、转移及基质金属铁蛋白酶-2、9活性的影响
蒿甲醚对人乳腺癌细胞的增殖抑制作用研究
PI3K信号通路抑制剂在人乳腺癌细胞侵袭的作用及可能机制
内皮-单核细胞激活多肽-Ⅱ增强血肿瘤屏障通透性的信号机制
信号分子PKC调控内皮-单核细胞激活多肽Ⅱ增强血肿瘤屏障通透性机制的研究
ROCK介导缓激肽开放SD大鼠血肿瘤屏障
EMAP-Ⅱ通过LKB1/AMPK/mTOR信号通路增加BTB的通透性