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痛情绪和痛记忆形成的细胞和分子机制
  • 所属机构名称：复旦大学
  • 会议名称：中华医学会疼痛分会第六届年会，2005，6， 17-20 青岛
  • 成果类型：会议
  • 相关项目：SNAP-25 相关蛋白(SIP30) 介导神经病理性疼痛的分子机制
同会议论文项目
SNAP-25 相关蛋白(SIP30) 介导神经病理性疼痛的分子机制
期刊论文 12 会议论文 13 著作 1
同项目会议论文
Involvement of ERK cascades in the Anterior Cingulate Cortex in Affective Pain.
NMDA-MAPK-CREB pathway mediated affective pain”
NMDA receptor in the ACC contributes to affective pain.
病理性疼痛的神经生物学机制
The Glycine site of NMDA Receptors in the Anterior Cingulate Cortex Contributes to Pain-Conditioned
MAPK in the anterior cingulate cortex contributes to pain-related aversion
SIP30 involves in neuropathic pain in rats
Is Functional State of Spinal Glia Involved in the Anit-allodynic and Anti-hyperalgesic Effects of E
ERK信号转导通路参与大鼠前扣带皮层痛相关厌恶情绪的形成
Is endogenous D-serine in the rostral anterior cingulated cortex necessary for pain-related negative
NMDAR-dependent AC-ERK-CREB signaling pathway in the anterior cingulate cortex contributes to pain-r
SIP30, a new MAPK downstream target in chronic constriction injuried neuropathic pain model in rats