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Diverse regulation of AKT and GSK-3 beta by O-GlcNAcylation in various types of cells
  • ISSN号：0014-5793
  • 期刊名称：FEBS Letters
  • 时间：2012.7.30
  • 页码：2443-2450
  • 相关项目：靶向抑制Dyrk1A阻断致病性tau形成及其在AD防治中的作用
作者： Shi, Jianhua|Wu, Shiliang|Dai, Chun-ling|Li, Yi|Grundke-Iqbal, Inge|Iqbal, Khalid|Liu, Fei|Gong, Cheng-Xin|
同期刊论文项目
靶向抑制Dyrk1A阻断致病性tau形成及其在AD防治中的作用
期刊论文 23 会议论文 4
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Developmental Regulation of Protein O-GlcNAcylation, O-GlcNAc Transferase, and O-GlcNAcase in Mammal
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Regulation of the alternative splicing of tau exon 10 by SC35 and Dyrk1A.
Cross talk between PI3K-AKT-GSK-3beta and PP2A pathways determines tau hyperphosphorylation.
Regulation of alternative splicing of tau exon 10 by 9G8 and Dyrk1A.
Cyclic AMP-dependent protein kinase regulates the alternative splicing of tau exon 10: a mechan
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Diverse regulation of AKT and GSK-3β by O-GlcNAcylation in various types of cells
Inhibition of protein synthesis alters protein degradation through activation of protein kinase B (A
Cyclic AMP-dependent protein kinase enhances SC35-promoted tau exon 10 inclusion.
Splicing factor SC35 promotes tau expression through stabilization of its mRNA.
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<span id="fck_dom_range_temp_1357348742869_974">SC35 promotes tau expression through
CREB regulates the expression of neuronal glucose transporter 3: a possible mechanism related to imp
蛋白激酶A催化亚基差异性地调节tau外显子10的可变剪接
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Dual-specificity tyrosine phosphorylation-regulated kinase 1A (Dyrk1A) enhances tau expression.
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