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Cyclic AMP-dependent protein kinase enhances SC35-promoted tau exon 10 inclusion.
  • ISSN号：0893-7648
  • 期刊名称：Mol Neurobiol.
  • 时间：2014
  • 页码：615-624
  • 相关项目：靶向抑制Dyrk1A阻断致病性tau形成及其在AD防治中的作用
作者： Qian W|Liu W|Tan X|Ding F|Gu X|Iqbal K|Gong C-X|Zu J|Liu F|
同期刊论文项目
靶向抑制Dyrk1A阻断致病性tau形成及其在AD防治中的作用
期刊论文 23 会议论文 4
同项目期刊论文
Developmental Regulation of Protein O-GlcNAcylation, O-GlcNAc Transferase, and O-GlcNAcase in Mammal
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Regulation of the alternative splicing of tau exon 10 by SC35 and Dyrk1A.
Diverse regulation of AKT and GSK-3 beta by O-GlcNAcylation in various types of cells
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Regulation of alternative splicing of tau exon 10 by 9G8 and Dyrk1A.
Cyclic AMP-dependent protein kinase regulates the alternative splicing of tau exon 10: a mechan
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Diverse regulation of AKT and GSK-3β by O-GlcNAcylation in various types of cells
Inhibition of protein synthesis alters protein degradation through activation of protein kinase B (A
Splicing factor SC35 promotes tau expression through stabilization of its mRNA.
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CREB regulates the expression of neuronal glucose transporter 3: a possible mechanism related to imp
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Dual-specificity tyrosine phosphorylation-regulated kinase 1A (Dyrk1A) enhances tau expression.
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