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Chronic cerebral hypoperfusion causes decrease of O-GlcNAcylation, hyperphosphorylation of tau
  • ISSN号：1663-4365
  • 期刊名称：Front Aging Neurosci
  • 时间：2014
  • 页码：10-10
  • 相关项目：靶向抑制Dyrk1A阻断致病性tau形成及其在AD防治中的作用
作者： Zhao, Yang|Gu, Jin-Hua|Dai, Chun-Ling|Liu, Qun|Iqbal, Khalid|Liu, Fei|Gong, Cheng-Xin|
同期刊论文项目
靶向抑制Dyrk1A阻断致病性tau形成及其在AD防治中的作用
期刊论文 23 会议论文 4
同项目期刊论文
Developmental Regulation of Protein O-GlcNAcylation, O-GlcNAc Transferase, and O-GlcNAcase in Mammal
Cyclic AMP-dependent protein kinase regulates 9G8-mediated alternative splicing of tau exon 10
Regulation of the alternative splicing of tau exon 10 by SC35 and Dyrk1A.
Diverse regulation of AKT and GSK-3 beta by O-GlcNAcylation in various types of cells
Cross talk between PI3K-AKT-GSK-3beta and PP2A pathways determines tau hyperphosphorylation.
Regulation of alternative splicing of tau exon 10 by 9G8 and Dyrk1A.
Cyclic AMP-dependent protein kinase regulates the alternative splicing of tau exon 10: a mechan
Cyclic AMP-dependent protein kinase regulates 9G8-mediated alternative splicing of tau exon 10.
Diverse regulation of AKT and GSK-3β by O-GlcNAcylation in various types of cells
Inhibition of protein synthesis alters protein degradation through activation of protein kinase B (A
Cyclic AMP-dependent protein kinase enhances SC35-promoted tau exon 10 inclusion.
Splicing factor SC35 promotes tau expression through stabilization of its mRNA.
TDP-43与神经退行性疾病
Regulation of alternative splicing of tau exon 10.
<span id="fck_dom_range_temp_1357348742869_974">SC35 promotes tau expression through
CREB regulates the expression of neuronal glucose transporter 3: a possible mechanism related to imp
蛋白激酶A催化亚基差异性地调节tau外显子10的可变剪接
Activation of Protein Phosphatase 2B and Hyperphosphorylation of Tau in Alzheimer's Disease
Dual-specificity tyrosine phosphorylation-regulated kinase 1A (Dyrk1A) enhances tau expression.
Truncation and activation of GSK-3beta by calpain I: a molecular mechanism links to tau hyperp