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G9a is essential for EMT-mediated metastasis and maintenance of cancer stem cell-like characters in
  • ISSN号：1949-2553
  • 期刊名称：Oncotarget
  • 时间：2015.3.30
  • 页码：6887-6901
  • 相关项目：肺慢性炎症恶性转化的分子机制
作者： Yadi Wu|Binhua P. Zhou|Zhiyuan Zhang|Jiong Deng|
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肺慢性炎症恶性转化的分子机制
期刊论文 15
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PI3K is required for the physical interaction and functional inhibition of NF-κB by β-catenin in col
Disrupting the Interaction of BRD4 with Diacetylated Twist Suppresses Tumorigenesis in Basal-like Br
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肿瘤代谢研究的新进展
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EGFR phosphorylates and inhibits lung tumor suppressor GPRC5A in lung cancer
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Gprc5a-deficiency confers susceptibility to endotoxin-induced acute lung injury via NF-κB pathway
Lung tumor suppressor GPRC5A binds EGFR and restrains its effector signaling
慢性阻塞性肺病的发生机制
Epithelial neoplasia coincides with exacerbated injury and fibrotic response in the lungs of Gprc5a-