位置：成果数据库 > 期刊 > 期刊详情页
氧化应激在慢性阻塞性肺疾病发生中的作用及防治策略
  • 期刊名称：国际呼吸杂志
  • 时间：0
  • 页码：-
  • 相关项目：肺慢性炎症恶性转化的分子机制
作者： 付玥川|丁婕|邓炯*|
同期刊论文项目
肺慢性炎症恶性转化的分子机制
期刊论文 15
同项目期刊论文
PI3K is required for the physical interaction and functional inhibition of NF-κB by β-catenin in col
G9a is essential for EMT-mediated metastasis and maintenance of cancer stem cell-like characters in
Disrupting the Interaction of BRD4 with Diacetylated Twist Suppresses Tumorigenesis in Basal-like Br
慢性炎症对肺癌发生发展的促进作用
肿瘤代谢研究的新进展
肺干细胞和肺癌干细胞的研究进展
EGFR phosphorylates and inhibits lung tumor suppressor GPRC5A in lung cancer
慢性阻塞性肺病与肺癌的相互关系
Gprc5a-deficiency confers susceptibility to endotoxin-induced acute lung injury via NF-κB pathway
Lung tumor suppressor GPRC5A binds EGFR and restrains its effector signaling
慢性阻塞性肺病的发生机制
Epithelial neoplasia coincides with exacerbated injury and fibrotic response in the lungs of Gprc5a-