位置：成果数据库 > 期刊 > 期刊详情页
PKM2 depletion induces the compensation of glutaminolysis through beta-catenin/c-Myc pathway in tumo
  • ISSN号：0898-6568
  • 期刊名称：Cellular Signalling
  • 时间：2014.11
  • 页码：2397-2405
  • 相关项目：乙酰化修饰在大肠癌细胞GRP78蛋白分泌中的调控作用
作者： Yang, Peng|Zhang, Lichao|Fan, Yongsheng|Li, Zhuoyu|
同期刊论文项目
乙酰化修饰在大肠癌细胞GRP78蛋白分泌中的调控作用
期刊论文 21
同项目期刊论文
Cell-surface GRP78 facilitates colorectal cancer cell migration and invasion
A millet bran-derived peroxidase inhibits cell migration by antagonizing STAT3-mediated epithelial-m
Overexpressed GRP78 affects EMT and cell-matrix adhesion via autocrine TGF-β/Smad2/3 signaling
GRP78 Secreted by Colon Cancer Cells Facilitates Cell Proliferation via PI3K/Akt Signaling
Direct contacts with colon cancer cells regulate the differentiation of bone marrow mesenchymal stem
GRP78 secreted by tumor cells stimulates differentiation of bone marrow mesenchymal stem cells to ca
The multifaceted regulation and functions of PKM2 in tumor progression
Pyruvate kinase M2 facilitates colon cancer cell migration via the modulation of STAT3 signalling
Glucose regulated protein 78 promotes cell invasion via regulation of uPA production and secretion i
Reconstructed mung bean trypsin inhibitor targeting cell surface GRP78 induces apoptosis and inhibit
Pyruvate kinase M2 accelerates pro-inflammatory cytokine secretion and cell proliferation induced by
Secreted pyruvate kinase M2 facilitates cell migration via PI3K/Akt and Wnt/β-catenin pathway in col
GRP78 is implicated in the modulation of tumor aerobic glycolysis by promoting autophagic degradatio
GRP78 enhances the glutamine metabolism to support cell survival from glucose deficiency by modulati
地皮菜中一种新型水应激蛋白基因的克隆表达及其抗人结肠癌细胞SW480的作用
以葡萄糖调节蛋白78（GRP78）为靶向的胰蛋白酶抑制剂抗肠癌效应研究
Gli1与β-catenin相互作用促进二者在结肠癌细胞中的协同核输入
丙酮酸激酶M1和M2的表达水平与结肠癌发展相关性研究