目的:从血液流变学改变探讨心病瘀血舌形成的病理机制。方法:随机选择72例心病瘀血舌患者、30例心病非瘀血舌患者、30例非心病瘀血舌患者与20例健康人,检测血液流变学各项指标。结果:心病瘀血舌患者血液存在"黏"、"浓"、"凝"、"聚"状态,且不同证型有不同的"黏"、"浓"、"凝"、"聚"倾向。血液凝聚性增高是不同血瘀证型的共同病理基础。阳虚血瘀证血液的"黏""聚"状况最为严重,痰瘀痹阻证和气滞血瘀证血液中的RBC聚集性增加明显;阳虚血瘀证和气虚血瘀证血液中的RBC变形能力下降显著;而阴虚血瘀证RBC聚集性增加和RBC变形能力减弱均很明显。痰瘀痹阻证、气滞血瘀证、阴虚血瘀证患者的血液处于"高浓"状态;气虚血瘀证和阳虚血瘀证的血液浓稠性改变并不明显。结论:心病瘀血舌的出现,是血液流变性异常改变至一定程度而导致舌微循环障碍的结果。血液流变学检测可为心病瘀血舌患者的诊断、辨证及疗效观察提供参考依据。
Objective:To explore the pathologic mechanism of blood-stasis-tongue subjects with cardiopathy(BSTC) from the change of hemorheology.Methods: 72 cases with BSTC,30 cases with non-blood-stasis-tongue subjects with cardiopathy(NBSTC) and 30 cases with blood-stasis-tongue subjects with non-cardiopathy(BSTNC) were observed at random,20 healthy cases as control.Hemorheology was examined.Results: The condition of "viscosity","thickness","coagulation" and "aggregation" existed in blood in patients of BSTC,and diff...