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Low-dose Endothelial Monocyte-activating Polypeptide-II Increases Permeability of Blood-tumor Barrie
Overexpression of Roundabout4 predicts poor prognosis of primary glioma patients via correlating wit
MiR-18aincreased the permeability of BTB via RUNX1 mediated down-regulation of ZO-1, occluding and c
C-terminus of Human BKca Channel Alpha Subunit Enhances the Permeability of the Brain Endothelial Ce
Functions for the cAMP/Epac/Rap1 Signaling Pathway in Low-Dose Endothelial Monocyte-Activating Polyp
Signal Mechanisms Underlying Low-Dose Endothelial Monocyte-Activating Polypeptide-II-Induced Opening
Roundabout 4 regulates blood-tumor barrier permeability through the modulation of ZO-1, Occludin, an
Roles of Serine/Threonine Phosphatases in Low-Dose Endothelial Monocyte-Activating Polypeptide-II-In
Low-Dose Endothelial Monocyte-Activating Polypeptide-II Increases Blood-Tumor Barrier Permeability b
Role of cAMP-dependent protein kinase A activity in low-dose endothelial monocyte-activating polypep
MiR-152 functions as a tumor suppressor in glioblastoma stem cellsby targeting Krüppel-like factor 4
miR-34c Regulates the Permeability of Blood-Tumor Barrier via MAZ-Mediated Expression Changes of ZO-
Roundabout4 suppresses glioma-induced endothelial cell proliferation, migration and tube formation i
Endothelial-monocyte-activating polypeptide II induces rat C6 glioma cell apoptosis via the mitochon
Low-dose endothelial monocyte-activating polypeptide-II increases permeability of blood–tumor barrie
Knockdown of long non-coding RNA HOTAIR inhibits malignant biological behaviors of human glioma cell
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