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中文摘要：
随着人口的老龄化，衰老及其相关疾病所引起的社会问题日益严重，出现寿命延长而生活质量下降的新问题，使有关衰老发生机制和防治措施的研究日显紧迫。本项目结合国内外有关衰老研究的最新进展、亟待解决的问题和申请者近年在突触可塑性（LTP/LTD）和氧自由基（ROS）两方面的研究工作基础，以ROS与突触可塑性年龄相关性改变的关系为切入点，综合运用电生理、免疫组化、分子生物学、质谱分析和行为学测试等技术，开展如下工作（1）
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Theaflavin ameliorates cerebral ischemia-reperfusion injury in rats through its anti -inflammatory e
Activation of phosphatidylinositol-linked novel D1 dopamine receptors inhibits high voltage activate
Effects of anthopleurin-Q on the intracellular free Ca2+ concentration in cultured rat cortical astr
Thrombin-induced microglial activation contributes to the degeneration of nigral dopaminergic neuron
Effect of human WEE1 and stem cell factor on human CD34+ umbilical cord blood cell damage induced by
Passive sensitization increases histamine-stimulated calcium signaling and NF-κB transcription activ
Activation of glycogen synthase kinase-3 inhibits long-term potentiation with synapse-associated imp
Inhibition of intracellular Ca(2+) release by a Rho-kinase inhibitor for the treatment of ischemic d
Activation of Phosphatidylinositol-linked Novel D(1) Dopamine Receptor Contributes to the Calcium Mo
Age-Related Alterations of Synapses in CA1 Stratum Radiatum and Spatial Learning Impairment in Rats.
Effect of progesterone on calcium activated potassium currents and intracellular calcium in guinea p
Differential Effects of Methionine and Cysteine Oxidation on [Ca(2+)] (i) in Cultured Hippocampal Ne
Puerarin facilitates Ca2+-induced Ca2+ release triggered by KCl-depolarization in primary cultured r
Chronic SKF83959 induced less severe dyskinesia and attenuated L-DOPA-induced dyskinesia in 6-OHDA-l
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