中文摘要：

分离和涉及发信号的赤霉素(气体) 的相关部件的功能的研究是重要的澄清 GA 工作的机制。关于 DELLA 的下游的部件的研究，表明小径的 GA 的关键因素，目前被限制。GASA (在 Arabidopsis 刺激 GA ) 家庭包含 GA 通常在 Arabidopsis thaliana 调整的 15 基因。所有 GASA 蛋白质在 C 终点包括 12 半胱氨酸在 N 终点和保存 GASA 域有劈得开的信号肽。 RT-PCR 分析表明 GASA4 和 GASA6 的表示是下面调整的，但是 GASA1 和 GASA9 在 DELLA 是起来调整的异种， gai-t6 和 rga-24 ，以及双异种，在于因此 GASA4 和 GASA6 被导致，但是 GASA1 和 GASA9 被外长的 GA ₃禁止。另外，另外的 GASA 基因的表示模式被 GA 和骆驼毛的织物调整，独立或合作地。大多数 GASA 基因在根，茎，叶子，花和发展中的 siliques 被表示。GASA6， GASA7， GASA8， GASA9， GASA10， GASA11 和 GASA12 的倡导者驾驶的 GUS 基因被用作记者，所有 GASA 基因在成长并且区分的器官和截去地区表示了，这被发现，建议在细胞生长和区别的这些基因的角色。这研究在 GA 和骆驼毛的织物发信号小径为 GASA 基因家庭的功能的学习提供了一个重要基础。

英文摘要：

Separation and functional research of related components involved in gibberellins （GAs） signaling are important to clarify the mechanism of GA functioning. Research on the downstream components of DELLA, the key factor of the GA signaling pathway, is limited at present. GASA （GA-Stimulated in Arabidopsis） family contains 15 genes usually regulated by GA in Arabidopsis thaliana. All GASA proteins have a cleavable signal peptide in N terminus and a conserved GASA domain including 12 cysteines in C terminus. RT-PCR analysis revealed that the expression of GASA4 and GASA6 were down-regulated, but GASA 1 and GASA9 were up-regulated in the DELLA mutants, gai-t6 and rga-24, as well as the double mutant, consisting with the results that GASA4 and GASA6 were induced, but GASA1 and GASA9 were inhibited by exogenous GA3. In addition, the expression patterns of other GASA genes were regulated by GA and ABA, separately or cooperatively. Most of GASA genes were expressed in roots, stems, leaves, flowers and developing siliques. GUS gene driven by the promoters of GASA6, GASA7, GASA8, GASA9, GASAIO, GASA11 and GASA12were used as reporters and it was found that all GASA genes expressed in the growing and differentiating organs and abscission zones, suggesting the role of these genes in cell growth and differentiation. This study provided an important basis for functional study of the GASA gene family in the GA and ABA signaling pathway.