中文摘要：

在在发情的心肌的房间损坏和 HSP 信使 rna 抄写之间的关系强调了烤 ers 用一个分光光度计，组织病理学说的技术，和荧光被学习量的逆转录 PCR (FQ RT-PCR ) 。结果证明肌酸激酶(CK ) 和 glutamic 焦葡萄酸氨基转移酶(通用终端) 的活动在坚持的热压力期间是感应的。心肌的纤维的主要损害是小粒的退化和坏死。组成或血缘的热的抄写震惊蛋白质 70 (HSC70 )信使 rna 是可变的。热的抄写震惊蛋白质 70 (HSP70 )信使 rna 在坚持的热应力期间显然被增加。结果证明 HSC70 信使 rna 抄写的变化与 HSC70 信使 rna 抄写的 CK，和水平的活动相反必须在坚持的热应力期间被用作心肌的房间损坏的一个符号。

英文摘要：

The relationship between myocardial cell damages and HSPs mRNA transcription in heat stressed broilers was studied using a spectrophotometer, the histopathological technique, and fluorescence quantitative reverse transcription PCR （FQ RT-PCR）. The results showed that the activities of creatine kinase （CK） and glutamic-pyruvic transaminase （GPT） were induction during the persistent heat stress. The major lesions of the myocardial fibers were granular degeneration and necrosis. The transcription of constitutive or cognate heat shock protein 70 （HSC70） mRNA was changeable. The transcription of heat shock protein 70 （HSPT0） mRNA was increased obviously in the course of persistent heat stress. The results showed that the change of HSC70 mRNA transcription was contrary to the activity of CK, and the level of HSC70 mRNA transcription must be used as a symbol of the myocardial cell damages in the course of persistent heat stress.