识别在装饰用的梨树米饭控制圆锥花序建筑学的量的特点 loci (QTL ) ,一张基因地图基于简单顺序重复(SSR ) 被构造生来的线(RIL ) 从在栽培变种 Xiushui 79 和 C Bao 之间的一个十字导出的标记和 254 recombinant。七个圆锥花序特点在三环境下面被调查。单个标记分析显示 27 个 SSR 标记的一个总数高度在所有三环境与圆锥花序特点被联系。单个地点解释的 phenotypic 变化的百分比从 2% ~ 35% 变化了。基于混合线性模型,为七个圆锥花序特点的 40 添加剂 QTL 的一个总数被合成间隔印射检测,向 1.2% 鈥 ? 解释 5% phenotypic 变化。在有超过10%解释 phenotypic 变化的 9 QTL 之中,二 QTL 每圆锥花序( NPB )为主要分支的数字,二为圆锥花序长度( PL ),二为小穗状花小穗密度( SD ),为第二等的分支的数字的每圆锥花序( NSB ),为第二等的分支分发密度( SBD )的,和为小穗状花小穗的数字的每圆锥花序( NS )分别地。qPLSD-9-1 和 qPLSD-9-2 是新奇多种的 loci,同时显示出 PL 和 SD 上的效果。qPLSD-9-1 为 PL 解释了 34.7% phenotypic 变化并且为 SD 的 25.4% phenotypic 变化分别地。qPLSD-9-2 分别地为 PL 和 SD 解释了 34.9% phenotypic 变化和 24.4% 。在两 QTL 的 C Bao 等位基因证明 PL 上的积极效果,和在两 QTL 的 Xiushui 79 等位基因在 SD 上显示出积极效果。圆锥花序特点的基因变化主要被归因于添加剂效果。QTL 脳环境相互作用不为添加剂 QTL 和添加剂脳添加剂 QTL 对是重要的。
To identify quantitative trait loci (QTLs) controlling panicle architecture in japonica rice, a genetic map was constructed based on simple sequence repeat (SSR) markers and 254 recombinant inbred lines (RILs) derived from a cross between cultivars Xiushui 79 and C Ban. Seven panicle traits were investigated under three environments. Single marker analysis indicated that a total of 27 SSR markers were highly associated with panicle traits in all the three environments. Percentage of phenotypic variation explained by single locus varied from 2% to 35%. Based on the mixed linear model, a total of 40 additive QTLs for seven panicle traits were detected by composite interval mapping, explaining 1.2%--35% ofphenotypic variation. Among the 9 QTLs with more than 10% of explained phenotypic variation, two QTLs were for the number of primary branches per panicle (NPB), two for panicle length (PL), two for spikelet density (SD), one for the number of secondary branches per panicle (NSB), one for secondary branch distribution density (SBD), and one for the num- ber of spikelets per panicle (NS), respectively, qPLSD-9-1 and qPLSD-9-2 were novel pleiotropic loci, showing effects on PL and SD simultaneously, qPLSD-9-1 explained 34.7% of the phenotypic variation for PL and 25.4% of the phenotypic variation for SD, respec- tively, qPLSD-9-2 explained 34.9% and 24.4% of the phenotypic variation for PL and SD, respectively. The C Bao alleles at the both QTLs showed positive effects on PL, and the Xiushui 79 alleles at the both QTLs showed positive effects on SD. Genetic variation of panicle traits are mainly attributed to additive effects. QTLx environment interactions were not significant for additive QTLs and additive x additive QTL pairs.