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中文摘要：
在盐度为12的海水中，添加不同浓度的CaCl2（2、4、6、8、10g／L）可对中华绒螯蟹肝胰腺中超氧化物歧化酶（S（）D）、碱性磷酸酶（AKP）、酸性磷酸酶（ACP）、过氧化氢酶（CAT）以及溶菌酶（LZM）的活性产生影响。结果表明：在实验海水中浸泡4h后，实验组与对照组河蟹肝胰腺中5种酶的活性随着CaCl2浓度的上升均出现了不同程度的降低，当CaCl2浓度为10g／L时，活性最低；ACP和AKP分别为对照组的56．72％和40．76％；SOD与CAT的活性在10g／L时为对照组的20．11％和7．32％；LZM的活性为对照组的60．14％；该结果说明：正常海水中CaCl2浓度的提高会导致河蟹体内自由基代谢的紊乱，从而影响机体的正常生理功能和免疫力。除LZM外，当CaCl2浓度为4g／L时，其它免疫酶的活性均表现出向下的拐点。因此，继续提高海水中的CaCI：浓度，将对河蟹成活率产生不利的影响。
英文摘要：
It is able to affect Chinese mitten crab pancreas liver SOD, alkaline phosphatase （AKP）, a cidie phosphatase （ACP）, catalase （CAT） and lysozyme （LZM） activity by adding different concentra tion of CaCl2（2, 4, 6, 8, 10g/L） in the sea water of salinity is12. The results showed that in the water 4 h, the experimental group and 5 control groups enzyme activity with the rising of the concentrations of CaC12 all appear different degrees of lower, and were all in the concentration of CaCl2 10 g/I. occurs the lowest; ACP and AKP respectively 56.72% and 40.76% in the control groups; The activity of SOD and CAT in 10 g/L for 20. 11% in control groups and 7.32%; LZM activity for 60. 14% in control groups; The results indicate that the increasing of CaC12 in the normal sea water will result in the in- creasing of the concentration of the free radical metabolism disorder and influence of the normal physio logical function and immunity, CaCl2 concentration for 4 g/L except LZM outside, the rest of the en- zyme activity is immune to show a downward inflection point, therefore continue to improve CaCl2 con- centration in sea water will affect the survival rate of crabs adversely.
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