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中文摘要：
选用感丝裂病的玉米自交系R08与抗丝裂病的自交系Es40组配F2群体共348个单株，构建了包含115个SSR标记的分子遗传连锁图谱，覆盖玉米基因组2178．6cM，平均图距为18．9cM。采用复合区间作图法，对F2：4家系丝裂病数据进行抗性QTL分析，共检测到12个QTL，分别位于第1、2、4、5和7染色体，贡献率为4．22％-37．95％。其中在第1、3染色体上检测到主效QTL，贡献率均大于30％，基因作用方式均为显性，其余10个QTL的作用方式多为加性或部分显性。
英文摘要：
Using 115 SSR markers and the F2 population consisting of 348 lines derived from the cross between maize （Zea mays L.） inbred lines R08 and Es40, a genetic linkage map associated with the resistance to silk cut was constructed. The genetic linkage map covered 2 178.6 cM of maize genome with an average mapping distance of 18.9 cM. Using the composite interval mapping （CIM） method, 12 QTLs controlling the resistance to silk cut were detected on chromosomes 1, 2, 4, 5, and 7. These QTLs explained phenotypic variances ranging from 4.22% to 37.95%. Among them, two major QTLs on chromosomes 1 and 3, which explained more than 30% of phenotypic variances, had dominance effect, whereas, the other 10 QTLs had partial dominance effect or additive effect.
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